Rol' sistemy renin-angiotenzin-al'dosteron i vazopressina v razvitii serdechnoǐ nedostatochnosti pri postoiannoǐ élektrostimuliatsii serdtsa.

Translated title of the contribution: The role of the renin-angiotensin-aldosterone system and vasopressin in the development of heart failure during continuous electric stimulation of the heart

V. I. Usov, I. B. Lishmanov, V. I. Chernov, B. R. Rezapov, S. D. Akhmedov, I. A. Travkov

Research Nuclear Reactor Center

Research output: Contribution to journal › Article › peer-review

Abstract

The status of the renin-angiotensin-aldosterone system and the time course of vasopressin levels were studied in patients with an implanted cardiostimulator. Activation of plasma renin due to renal hypoperfusion in an inadequate stimulation was demonstrated to be followed by a rise in aldosterone and vasopressin concentrations and by an increase in blood volumes. Activation of angiotensin II and vasopressin resulted in higher peripheral resistance. Altered humoral regulation of the circulation retained 6-12 months after cardiostimulator implantation, by maintaining myocardial overload with volume and resistance.

Translated title of the contribution	The role of the renin-angiotensin-aldosterone system and vasopressin in the development of heart failure during continuous electric stimulation of the heart
Original language	Russian
Pages (from-to)	52-55
Number of pages	4
Journal	Kardiologiya
Volume	30
Issue number	1
Publication status	Published - Jan 1990

ASJC Scopus subject areas

Cardiology and Cardiovascular Medicine

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title = "Rol' sistemy renin-angiotenzin-al'dosteron i vazopressina v razvitii serdechnoǐ nedostatochnosti pri postoiannoǐ {\'e}lektrostimuliatsii serdtsa.",

abstract = "The status of the renin-angiotensin-aldosterone system and the time course of vasopressin levels were studied in patients with an implanted cardiostimulator. Activation of plasma renin due to renal hypoperfusion in an inadequate stimulation was demonstrated to be followed by a rise in aldosterone and vasopressin concentrations and by an increase in blood volumes. Activation of angiotensin II and vasopressin resulted in higher peripheral resistance. Altered humoral regulation of the circulation retained 6-12 months after cardiostimulator implantation, by maintaining myocardial overload with volume and resistance.",

author = "Usov, {V. I.} and Lishmanov, {I. B.} and Chernov, {V. I.} and Rezapov, {B. R.} and Akhmedov, {S. D.} and Travkov, {I. A.}",

year = "1990",

month = jan,

language = "Русский",

volume = "30",

pages = "52--55",

journal = "Kardiologiya",

issn = "0022-9040",

publisher = "Media Sfera",

number = "1",

}

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T1 - Rol' sistemy renin-angiotenzin-al'dosteron i vazopressina v razvitii serdechnoǐ nedostatochnosti pri postoiannoǐ élektrostimuliatsii serdtsa.

AU - Usov, V. I.

AU - Lishmanov, I. B.

AU - Chernov, V. I.

AU - Rezapov, B. R.

AU - Akhmedov, S. D.

AU - Travkov, I. A.

PY - 1990/1

Y1 - 1990/1

N2 - The status of the renin-angiotensin-aldosterone system and the time course of vasopressin levels were studied in patients with an implanted cardiostimulator. Activation of plasma renin due to renal hypoperfusion in an inadequate stimulation was demonstrated to be followed by a rise in aldosterone and vasopressin concentrations and by an increase in blood volumes. Activation of angiotensin II and vasopressin resulted in higher peripheral resistance. Altered humoral regulation of the circulation retained 6-12 months after cardiostimulator implantation, by maintaining myocardial overload with volume and resistance.

AB - The status of the renin-angiotensin-aldosterone system and the time course of vasopressin levels were studied in patients with an implanted cardiostimulator. Activation of plasma renin due to renal hypoperfusion in an inadequate stimulation was demonstrated to be followed by a rise in aldosterone and vasopressin concentrations and by an increase in blood volumes. Activation of angiotensin II and vasopressin resulted in higher peripheral resistance. Altered humoral regulation of the circulation retained 6-12 months after cardiostimulator implantation, by maintaining myocardial overload with volume and resistance.

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